There is evidence of acute parenchymal cytogenic edema or post ischemic leukomalacia matching any arterial afferent perfusion zone, any watershed zone, nor any efferent venous egress zone.

There is evidence of hyperintense, acute, intraluminal arterial thrombosis in a major stem, trunk, division or pial artery perfusion zone.

There is evidence of hyperintense, acute, intraluminal arterial thrombosis in a recognizable perforator, choroidal, or deep subcortical penetrating artery/arteries.

There is evidence of hypo or hyperintensity within the deep medullary veins, or the deep venous system to suggest congestion, stasis, thrombosis, or increased (high flow) medullary venous hyperemia

There is hyperintensity within the cortical ribbon consistent with sequestered infarction (laminar necrosis)

There is hyperintensity within the centronuclear structures consistent with chronic low grade ischemic changes

There is enlargment of the Virchow-Robin spaces surrounding either the perforating arteries or the penetrating arteries.

There is luminal abnormal isointensity (lack of flow void) or hyperintensity within one or more dural sinuses (delta sign) indicative of acute thrombus on the noncontrast T1.

There is evidence of hyper or isointensity within one or more cortical or central veins on noncontrast T1.

There is evidence of dilatation of the intraorbital veins, and optic hydrops (dilated CSF space around the optic nerves) on pre or post contrast T1 MR.

There is evidence of abnormal ventriculomegaly consistent with early hydrocephalus, pseudotumor, or PRES.

No other findings abnormalities are noted.