Section 1

Submit Findings

Case210a

Findings

Observation: Evidence of a Dural Sinus Thrombosis or Co-Morbid Cause for Thrombosis

​There is evidence of acute thrombosis of any dural sinus or the torcular herophile.

​There is evidence of acute thrombosis of the jugular fossa segment of the sigmoid sinus (possible “jugular fossa syndrome”).

​There is evidence of acute thrombosis of either high cervical internal jugular veins.

​There is evidence of a straight sinus thrombosis.

​There is evidence of aggressive otomastoid sinus disease, which could lead to internal jugular vein or sigmoid or transverse sinus thrombosis.

​There is evidence of cavernous sinus thrombosis.

​There is evidence of aggressive sphenoid sinus or retropharyngeal infectious disease, which could lead to cavernous sinus thrombosis.

​There is evidence of dilatation of the orbital veins (optic hydrops).

Observation: Evidence of a Recognizable Cortical Vein Thrombosis or Co-Morbid Cause for Thrombosis

​There is evidence of supratentorial cortical vein thrombosis or cytogenic edema corresponding to a recognizable cortical vein (i.e. a cerebral venous vector).

​There is evidence of infratentorial cortical vein thrombosis or cytogenic edema corresponding to a recognizable cortical vein (i.e. a posterior fossa venous vector).

​There is evidence of frontal or frontoethmoid sinusitis, which can cause frontal vein thrombophlebitis.

​There is evidence of deep central vein (ICV, septal vein, thalamostriate) or vein of Galen possibly including the straight sinus thrombosis.

​There is evidence of basal vein of Rosenthal thrombosis.

​There is evidence of invasive or allergic (AFS) nasopharyngeal fungal disease, which could lead to cavernous sinus thrombosis.

​There is evidence of post surgical vein sacrifice.

Observation: Evidence of Secondary Effects of Venous Hypertension

​There is evidence of recent intracranial parenchymal (intra axial) hemorrhage in a venous egress zone.

​There is evidence of recent intracranial subarachnoid/cisternal hemorrhage.

​There is evidence of subpial prepontine venous hemorrhage.

​There is evidence of early hydrocephalus.

​There is evidence of pseudotumor cerebri.

Other

​Other findings are present consistent with a disorder other than CVT.