Hyperacute Seizure 10 - Clinical Case Summary
Hyperacute Seizure 10 - Clinical Case Summary
Hyperacute Seizure 10 - Clinical Case Summary
SummaryHistory
29 yo male with history of nocturnal seizures as a child. Now presents with transient dyscognition and automtisms.
Exams Performed
MR Coronal T2-w spin echo and MR FLAIR; MR diffusion
Prior available imaging reports
CT Head and T1-w imaging is not available
MR Coronal T2-w spin echo
1. There is evidence of bilateral, hyperacute, post-seizure tissue injuries (edema) involving all of the left hippocampus but only the tail of the right hippocampus.
MR diffusion (DWI)
1. There are bilateral hyperacute, post-seizure changes affecting most of the left hippocampus, but only the tail of the right hippocampus.
MR Susceptibility (SWI) is not available
MR Coronal T2-w spin echo
1. There is evidence of bilateral, hyperacute, post-seizure tissue injuries (edema) involving all of the left hippocampus but only the tail of the right hippocampus.
MR diffusion (DWI)
1. There are bilateral hyperacute, post-seizure changes affecting most of the left hippocampus, but only the tail of the right hippocampus.
MR Susceptibility (SWI) is not available
Overall impression
1. There is a clinical history of possible seizure related symptoms but no clear clinical seizure manifestations. The MR seizure protocol imaging demonstrates clear evidence of recent seizure activity affecting all parts of the left hippocampus. There is cytogenic edema, effacement of internal and external white matter tracts in the head body and tail. The right hippocampus is normal in the head and body but has focal post seizure injury in only the tail portion of the hippocampus. This is not uncommon. Therefore, careful analysis of all segments of each hippocampus is necessary to detect all the pertinent post ictal injuries.