There is hyperemic pial circulation without contrast leak consistent with dysautoregulation post seizure.

There is reduced or absent post contrast signal intensity within brain parenchyma to suggest oligemic stroke (arterial, venous, or transcapillary).

There is thrombus in either dural sinus(es) and or cortical veins (CVT), as a cause for edema and seizure.

There is contrast enhancement (contrast leak) which follows the parenchymal Virchow-Robin spaces surrounding the small metarterioles consistent with angiitis. This may create a patterns suggesting small nodule when viewed in axial plane.

There is evidence of unexpected regional cortical vein enlargement (arterialization of othersise NL veins), consistent with pial AVM, or dural AV fistula plus dural sinus stenosis/thrombosis.

There is evidence of skull base destruction with enhancement (infection/tumor) leading to cavernous or dural sinus thrombosis.

There is abnormal intraaxial parenchymal enhancement to suggest an infiltrative neoplastic tumor or encephalitis.

There is evidence of tumoral regional hypervascularity with or without neoplastic type of AV shunting.

There is abnormal extraaxial enhancement to suggest an aggressive meningeal process (infectious or neoplastic).

Other significant findings are present.