Info Images Findings Impression Reco/Acuity Case Images View Images / Launch Visage Case Notes History 66yo male prior history of seizures, hypertension, and prior stroke who fell at home. Pt did lose consciousness and was amnestic for event. Exam Noncontrast head CT Prior Study none Dicom View Reference Material
Section 1 Submit Findings CB1085 Findings Head CT There is evidence of subperiosteal hemorrhage along either the inner or outer calvarial surface. Yes No There is evidence of epidural hemorrhage (acute-meningeal artery bleed vs delayed-diploic venous bleed). Yes No There is evidence of intradural hemorrhage (usually Peds patients). Yes No There is evidence of subdural hemorrhage. Yes No There is evidence of hemorrhage into CSF spaces including subarachnoid, intraventricular, or subpial spaces. Yes No There is evidence epidural/sub periosteal/parenchymal blood outside dural sinuses, especially for injury adjacent to the superior sagittal or transverse sinuses. Yes No There is evidence of epidural blood adjacent to fx near meningeal arteries. Yes No Mixed-age CNS injuries in the absence of clear history in a child is very suspicious for NAI. Yes No Assess for intracranial/intraaxial or intraventricular post traumatic injuries There is evidence of intraaxial direct impact injuries: brain contusion or superficial gliding contusions (over orbital roof or petrous bone). Yes No There is evidence of intraaxial/extraaxial direct impact injuries (coup event) and opposite side (contre coup event), which can include intraventricular hemorrhage. Yes No There is evidence of brain laceration from missile type penetrating injuries. Yes No There is evidence of intraaxial (non-direct impact acceleration-deceleration) brain shear injuries or diffuse axonal injuries (i.e. DAI) causing microhemorrhages at the gray-white junction or along the major forceps of the corpus callosum. Yes No There is evidence of microhemorrhage from arterial microvascular shear effect (from acceleration-deceleration vector) in corpus callosum. Yes No There is evidence of microhemorrhage from arterial microvascular shear effect (from acceleration-deceleration vector) in the BG/thalamus, or brainstem. Yes No There is evidence of venous tether avulsion injuries, possibly with brain laceration (linear parenchymal hemorrhage). Yes No Assess for focal, regional, or diffuse brain swelling There is evidence of any focal intracranial post traumatic mass (or hydrocephalus) producing brain herniation. Yes No There is evidence of regional brain swelling from dysautoregulation and expanded intravascular pool, but without apparent brain edema. Yes No There is evidence of intercurrent PCA or circummesencephalic artery stroke(s) from tentorial herniation. Yes No There is evidence of intercurrent ACA stroke from subfalcine shift. Yes No There is evidence of intercurrent ICA stroke from traumatic injury or from uncal brain herniation compressing across the interclinoidal ligament. Can also be caused by choking compressing ICA in neck in NAI. Yes No There is evidence of an extraaxial hemorrhage adjacent to, and displacing or occluding a dural sinus. Yes No There is evidence of focal posterior temporal intra or extraaxial mass effect (usually associated with temporal bone fracture) compressing or occluding the V of Labbe’ and/or transverse sinus. Yes No There is evidence of global (diffuse) cytogenic edema likely related to concurrent apneic episode with cytotoxic brain edema. Yes No Assess for arterial or venous injury on CTA (or MRA) There is evidence of major proximal or pial arterial post traumatic dissection. Yes No N/A There is evidence of pial or meningeal arterial entrapment in a fracture. Yes No N/A There is evidence of major arterial post traumatic injury producing a pseudoaneurysm. Yes No N/A There is evidence of major arterial post traumatic injury producing a high or low flow AV fistula. Yes No N/A There is evidence of deformity (external compression) or thrombosis of a dural sinus. Yes No N/A There is abnormal capillary leak in areas of lost blood brain barrier, abnormal venocapillary pool density, or actual abnormal contrast enhancement if a focal lesion. Yes No N/A Other There is evidence of concurrent abnormalities not likely related to recent brain injury. Yes No