There is primary sinonasal, bone or skull base rather than a primary orbital disease process that might be causative and producing the patient’s signs and/or symptoms related to the orbit.

The pre-septal soft tissues including the lacrimal sac are abnormal.

The lacrimal gland is abnormal.

The bones and bony margins of the orbit including all component parts of the sphenoid bone are abnormal.

There is a structural abnormality or infiltrating process of the extraconal or intraconal compartments of the orbit.

The extraocular muscles appear to be swollen or otherwise abnormal.

The orbital apex and superior orbital fissure are infiltrated, edematous or otherwise abnormal.

There is enlargement and/or thrombosis of the superior, inferior or other orbital veins.

There is evidence of cavernous sinus thrombosis and/or inflammatory morphologic features in the cavernous sinus or para-cavernous region.

Proptosis is present.

The optic nerve is stretched in appearance.

The posterior aspect of the globe is tented in appearance.

Signs of tension orbit are present.

The uveal and scleral components of the globe are swollen or enhancing abnormally.

There is a hemorrhage or other abnormality causing a detachment of the choroid, hyaloid membrane and/or retina.

The optic disc region is abnormally swollen or enhancing.

The vitreous body is abnormal in appearance.

The anterior segment structures including the lens are abnormal in appearance.

Signs of endophthalmitis are present.

The optic nerves and/or optic sheaths appear abnormal in size.

The optic nerves appear otherwise abnormal.

There is abnormal enhancement of the optic nerves and/or of the optic sheath.

There is a structural abnormality or infiltrating process (especially around the optic sheath) of the intraconal compartment of the orbit.

There is a compressive lesion or infiltrative process present anywhere along the course of the optic nerves, optic chiasm or optic tracts before entering the brain.

There is a compressive lesion or infiltrative process present in the cavernous sinus or para-cavernous area.

There is evidence of an aneurysm or carotid cavernous fistula present.

There is abnormal meningeal or other enhancement of the optic chiasm, optic nerves or other structures in the suprasellar and/or chiasmatic cistern.

There is generalized abnormal meningeal enhancement.

There are intra-axial or extra-axial abnormalities of the brain that might be related to the orbital pathology.

There is evidence of obstructive or communicating hydrocephalus.