There is evidence of focal parenchymal hemorrhage.

There is evidence of intra ventricular bleeding.

There is evidence of concurrent parenchymal plus SAH to suggest a sentinel aneurysm bleed.

There is evidence of only trace amount of blood in occipital horns, posterior intrasylvian sulci, or foramen cecum part of the interpeduncular fossa.

There is evidence of intercurrent obstructive hydrocephalus.

There is evidence of  subarachnoid, pial or subpial bleed adjacent to cortical veins or dural sinuses.

There is evidence of SAH near falx (possibly post traumatic in origin).

There is evidence of vertex sulcal blood (possibly reversible vasoconstrictive vascular disorders).

There is evidence of focal cytogenic edema related to intercurrent hemorrhagic stroke.

There is evidence of thrombosed pial artery/vein as a cause for intraaxial hemorrhage.

There is evidence of thrombosed vascular malformation.

There is evidence of focal subarachnoid blood, possibly from saccular aneurysm associated with an AVM.

There is evidence of hemorrhagic met or other tumor as basis for SAH.

There is evidence of multicentric bleeding sites consistent with coagulopathy as cause for bleeding.

There is evidence of visibly enlarged pial or deep veins representing the venous egress of a pial AVM.

There is evidence of visibly enlarged dural sinuses, or enlarged orbital/pial veins with proptosis representing the venous egress of a dural AVF.

There is evidence of markedly enlarged pial or deep veins representing the venous egress of a direct AV fistula.

There is evidence of markedly enlarged internal cerebral veins and vein of Galen consistent with Galenic AV malformation.

Other abnormality.