There is soft tissue swelling of the scalp.

There is evidence of a fracture-linear/mixed, nondisplaced/displaced/depressed/complex calvarial fracture with or without diastasis.

There is evidence of a fracture crossing any of the meningeal artery or major dural sinuses sites.

There is evidence of a calvarial fracture extending into or from the intracranial surface of any of the paranasal sinuses, temporal bone /sigmoid plate/ lesser sphenoid wing or optic canal with or without intracranial gas.

There is evidence of concurrent abnormalities not likely related to recent brain injury.

There is evidence of subperiosteal/subgaleal hemorrhage along either the inner or outer calvarial surface.

There is evidence of epidural/subdural/intradural/ subarachnoid hemorrhage.

There are mixed-age CNS injuries.

There is evidence of  intraaxial direct impact injuries: brain contusion or superficial gliding contusions /coup opposite side (counter coup event), / intraventricular hemorrhage.

There is evidence of brain laceration from missile type penetrating injuries.

There is evidence of intraaxial (non-direct impact acceleration/deceleration) brain shear injuries or diffuse axonal injuries (i.e. DAI) arterial microvascular shear effect causing microhemorrhages at the gray-white junction or along the major forceps of the corpus callosum.

There is evidence of venous tether avulsion injuries, possibly with brain laceration (linear parenchymal hemorrhage).

There is evidence of any brain herniation.

There is evidence of regional brain swelling from dysautoregulation and expanded intravascular pool, but without apparent brain edema.

There is evidence of intercurrent ICA/ACA/PCA or circummesencephalic artery stroke(s) from herniation or choking.

There is evidence of global (diffuse) cytogenic edema likely related to concurrent apneic episode with cytotoxic brain edema.

There is evidence of major arterial post traumatic dissection.

There is evidence of pial or meningeal arterial entrapment in a fracture.

There is evidence of major arterial post traumatic injury producing a pseudoaneurysm.

There is evidence of major arterial post traumatic injury producing a high or low flow AV fistula.

There is evidence of deformity (external compression) or thrombosis of a dural sinus.

There is evidence of concurrent abnormalities not likely related to recent brain injury.