MR Diffusion
Claim CME CreditPOINT OF CARE INFORMATION
This CME activity consists of the student reviewing the video of the professor reviewing the case as well as the associated DICOM image set related to the case in question.
Learning Objectives
As a result of participation in this activity, participants should be able to:
- Provide improved patient care.
- Greater knowledge of the imaging characteristics of the patient's disease.
- Understand a better approach to interpretation of studies.
Faculty Disclosure
Mehmet Albayram, MD, Ivan Davis, MD, Mariam Hanna, MD, Anthony Mancuso, MD, Ronald Quisling, MD, Dhanashree Rajderkar, MD, Priya Sharma, MD, Roberta Slater, MD and Joann Stamm, MBA have disclosed that they have no relevant financial relationships. No one else is a position to control content have any financial relationship to disclose.
CME Advisory Committee Disclosure:
Conflict of interest information for the CME Advisory Committee members can be found on the following website: https://cme.ufl.edu/disclosure/.
Continuing Medical Education Credit
Accreditation: The University of Florida College of Medicine is accredited by the Accreditation Council for Continuing Medical Education (ACCME) to provide continuing medical education for physicians.
Credit: The University of Florida College of Medicine designates this enduring material for a maximum of 0.5 AMA PRA Category 1 Credits. Physicians should claim only the credit commensurate with the extent of their participation in the activity.
CA0387-MR Diffusion
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CA0387-MR Diffusion
Case ReportHistory
Exam
Prior Study
1. Prior occlusion of the posterior M3 trunk off the superior division Lt MCA, which has good pial collateral preventing extension of the stroke to most of the posterior superior division perfusion zone.
2. There is acute thrombus in single artery (artery to the central sulcus). The affected perfusion zone for this artery is much smaller than its’ full (normal) perfusion territory, which indicates there has been substantial retrograde pial collateralization from the A4 (ACA) pial vessels. However, there remains a small pial collateral gap in the mid-insula and the adjacent frontoparietal opercular cortex. In this case, the affected area includes the tongue part of the primary facial motor area, which correlates with the patients presenting symptom of dysarthria.
3. The improved CT density in the venocapillary pool is a good indication that the extent of final stroke-zone will remain quite small.
Findings
MR diffusion
Both the DWI and ADC maps are positive for a small stroke in mid-insula and part of the adjacent intrasylvian frontoparietal operculum. This matches the expected perfusion zone of the proximal part of the artery to the central sulcus vascular territory. Thus, the distal lateral sylvian cortical part of the superior division MCA’s expected perfusion zone is adequately perfused by retrograde ACA (A4) pial collaterals. The site of positive diffusion likely affects the tongue portion of the motor cortex accounting for the patient’s acute dysarthria.
As expected, there is no positive diffusion signal in the chronic stroke in the right posterior insular region.
