Hemorrhage - Case 9 MR T2-w
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This CME activity consists of the student reviewing the video of the professor reviewing the case as well as the associated DICOM image set related to the case in question.
Learning Objectives
As a result of participation in this activity, participants should be able to:
- Provide improved patient care.
- Greater knowledge of the imaging characteristics of the patient's disease.
- Understand a better approach to interpretation of studies.
Faculty Disclosure
Mehmet Albayram, MD, Ivan Davis, MD, Mariam Hanna, MD, Anthony Mancuso, MD, Ronald Quisling, MD, Dhanashree Rajderkar, MD, Priya Sharma, MD, Roberta Slater, MD and Joann Stamm, MBA have disclosed that they have no relevant financial relationships. No one else is a position to control content have any financial relationship to disclose.
CME Advisory Committee Disclosure:
Conflict of interest information for the CME Advisory Committee members can be found on the following website: https://cme.ufl.edu/disclosure/.
Continuing Medical Education Credit
Accreditation: The University of Florida College of Medicine is accredited by the Accreditation Council for Continuing Medical Education (ACCME) to provide continuing medical education for physicians.
Credit: The University of Florida College of Medicine designates this enduring material for a maximum of 0.5 AMA PRA Category 1 Credits. Physicians should claim only the credit commensurate with the extent of their participation in the activity.
CB1717-Hemorrhage - Case 9 MR T2-w
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CB1717-Hemorrhage - Case 9 MR T2-w
Case ReportHistory
Exam
Prior Study
1. There is evidence of concurrent subarachnoid hemorrhage with subarachnoid clot concentrated in the proximal segment of the sylvian fissure and right lateral suprasellar space plus a large intraparenchymal hematoma in the adjacent mesial right temporal lobe. This complex of SAH & adjacent intraaxial is consistent with a sentinel hemorrhage condition where pial adhesions from prior hemorrhage (which this patient had from the left MCA aneurysm bleed) cause the hemorrhage direction to bleed into brain and subarachnoid space at the same time.
2. There is minimal to moderate external hydrocephalus and papiiledema.
3. There is early downward uncal herniation compressing the right cerebral peduncle.
CT head: CTA
1. There is no residual aneurysm in the left MCA treatment area.
2. There is a right P-com saccular aneurysm with a relatively wide neck for fundal size. There is no vasospasm nor incorporation of the parent artery in the aneurysm neck. It has a small apical daughter aneurysm.
3. There is a small basilar apex pre-aneurysm.
MR T1-w pre contrast
1. The right temporal hematoma is virtually isointense to brain indicating it is in a hyperacute to acute timeframe (2 hrs to 2 days). The existing vasogenic perilesional edema is hypointense on T1 and surrounds the parenchymal hematoma.
2. There is a right posterior saccular aneurysm. It is not as well seen on MRA than on the CTA. There is no residual left MCA aneurysm, and the basilar tip pre-aneurysm is not evident.
3. The large right temporal mass reduces venocapillary filling, But this could be just delay in filling and not necessarily infarction.
MR T1-w post contrast/MRA
1. There is no residual aneurysm in the left MCA treatment area.
2. The active P-com aneurysm is present but is detail concerning the aneurysm is clearer on the CTA.
Findings
T2-w spin echo
There is an uncompacted hematoma in the right temporal lobe, as described above. There is possibly early deoxy Hgb forming the mesial aspect of the hematoma, but best estimate is that this hematoma is 12 to 24 hours in age.
There obstructive external minimal to moderate acute hydrocephalus (grad 1.4/4). A left frontal ventriculostomy has been placed with the tip free within the body of the lateral ventricle. Residual blood products remain the trigones and occipital horns bilaterally.
