AVM Case 4, CTA
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This CME activity consists of the student reviewing the video of the professor reviewing the case as well as the associated DICOM image set related to the case in question.
Learning Objectives
As a result of participation in this activity, participants should be able to:
- Provide improved patient care.
- Greater knowledge of the imaging characteristics of the patient's disease.
- Understand a better approach to interpretation of studies.
Faculty Disclosure
Mehmet Albayram, MD, Ivan Davis, MD, Mariam Hanna, MD, Anthony Mancuso, MD, Ronald Quisling, MD, Dhanashree Rajderkar, MD, Priya Sharma, MD, Roberta Slater, MD and Joann Stamm, MBA have disclosed that they have no relevant financial relationships. No one else is a position to control content have any financial relationship to disclose.
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Conflict of interest information for the CME Advisory Committee members can be found on the following website: https://cme.ufl.edu/disclosure/.
Continuing Medical Education Credit
Accreditation: The University of Florida College of Medicine is accredited by the Accreditation Council for Continuing Medical Education (ACCME) to provide continuing medical education for physicians.
Credit: The University of Florida College of Medicine designates this enduring material for a maximum of 0.5 AMA PRA Category 1 Credits. Physicians should claim only the credit commensurate with the extent of their participation in the activity.
CB1674-AVM Case 4, CTA

CB1674-AVM Case 4, CTA
Case ReportHistory
Exam
Prior Study
Findings
Findings
There is evidence of multicentric AV angiomatous transformations (a variant of arteriovenous malformation) located in three separate areas. The largest area of involvement is in the right posterior frontal centrum semiovale. The other two are small lesions: one is along the posterior pulvinar and the other is in the left lateral basifrontal cortex. A distinct nidus is absent in each lesion. Rather the afferent microfistula arteries appear dispersed within focal areas of brain parenchyma; the dilated efferent veins drain to either deep medullary venous system or to adjacent pial veins.
The largest site of the angiomatous AV transformation is located in the right mid, high convexity, frontal centrum semiovale. It is centered deep to the right premotor area, but not a usual eloquent site. This vascular lesion has diameter measures approximately 11 x 25 mm's. The afferent vessels arise from superior division MCA pial penetrating branches. The efferent vessels drain to the to the pial veins; there is no deep medullary drainage. The AV shunt volume is low. There is no evidence of flow related aneurysms arising from the afferent parent arteries. There is no apparent evidence of efferent outflow stenosis. There is no evidence of venous aneurysms. The Spetzler-Martin category is estimated at 1. Angiomatous AV transformations, as in this case, represent focal-micro-AV-fistulae. This type of AV-shunting vascular malformation draws from pial and their penetrating vessels. They represent a variant of AVM without definable nidus, but with AV shunting that is less aggressive than either a direct AV fistulae or a typical AVM.
The second site of focal angiomatous AV transformation is located in the left basifrontal area. This vascular malformation draws from basifrontal pial vessels; it does not involve an eloquent site. This lesion has diameter measures approximately 8 mm's. The efferent vessels drain to local pial veins. The AV shunt volume is low. There is no evidence of flow related aneurysms arising from the afferent parent arteries, nor evidence of efferent outflow stenosis, nor evidence of venous aneurysms. The Spetzler-Martin category is estimated at 1.
The third site of focal angiomatous AV transformation is located in the left mesial hippocampus/lateral geniculate nucleus area. This vascular malformation draws from the mesial PCA arteries. This lesion has diameter measures approximately 8-9 by 4 mm's. The efferent vessels drain to local pial veins flowing into the left BVR. There is no evidence of flow related aneurysms arising from the afferent parent arteries, nor evidence of efferent outflow stenosis, nor evidence of venous aneurysms. The AV shunt volume is low. The Spetzler-Martin category is estimated at 1.