Osteomyelitis, foot, MR
Claim CME CreditPOINT OF CARE INFORMATION
This CME activity consists of the student reviewing the video of the professor reviewing the case as well as the associated DICOM image set related to the case in question.
Learning Objectives
As a result of participation in this activity, participants should be able to:
- Provide improved patient care.
- Greater knowledge of the imaging characteristics of the patient's disease.
- Understand a better approach to interpretation of studies.
Faculty Disclosure
Mehmet Albayram, MD, Ivan Davis, MD, Mariam Hanna, MD, Anthony Mancuso, MD, Ronald Quisling, MD, Dhanashree Rajderkar, MD, Priya Sharma, MD, Roberta Slater, MD and Joann Stamm, MBA have disclosed that they have no relevant financial relationships. No one else is a position to control content have any financial relationship to disclose.
CME Advisory Committee Disclosure:
Conflict of interest information for the CME Advisory Committee members can be found on the following website: https://cme.ufl.edu/disclosure/.
Continuing Medical Education Credit
Accreditation: The University of Florida College of Medicine is accredited by the Accreditation Council for Continuing Medical Education (ACCME) to provide continuing medical education for physicians.
Credit: The University of Florida College of Medicine designates this enduring material for a maximum of 0.5 AMA PRA Category 1 Credits. Physicians should claim only the credit commensurate with the extent of their participation in the activity.
CB1132-Osteomyelitis, foot, MR
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CB1132-Osteomyelitis, foot, MR
Case ReportHistory
Exam
Findings
Findings
Soft tissue defect along the plantar aspect of the foot at the level of the first metatarsophalangeal joint. This extends down to the underlying flexor hallucis longus tendon sheath. There is surrounding enhancing edema, consistent with cellulitis.
Fluid and peripheral enhancement about the flexor hallucis longus tendon, consistent with infectious tenosynovitis, extending from the level of the great toe interphalangeal joint to the level of the mid first metatarsal.
Increased T2 signal within the lateral hallux sesamoid with associated focal loss of T1 signal, directly adjacent to the soft tissue inflammation discussed above, consistent with osteomyelitis. There is no discernible enhancement in the region of bone marrow edema in the lateral hallux sesamoid, suggesting loss of bone viability.
There is a peripherally enhancing fluid collection along the dorsum of the foot, communicating with the first toe metatarsophalangeal joint (best identified on the sagittal plane), consistent with septic arthritis.
Nonenhancing edema along the dorsum of the foot, likely transudative edema.
Changes of diabetic myopathy.
Impression
Osteomyelitis of the lateral hallux sesamoid with possible loss of bone viability.
First metatarsophalangeal septic arthritis.
