Head Trauma - MR susceptibility (SWI)
Head Trauma - MR susceptibility (SWI)
Search Pattern Assist ?Exam
Purpose
2. SWI is very effective in evaluating deep venous drainage which reflects transcapillary perfusion. Therefore, it can assess both hyperemia (increased flow) or hypoperfusion (decreased flow).
Findings
MR susceptibility (SWI)
There is SWI evidence of abnormal susceptibility artifact associated with extraaxial blood products. [Yes/No]
There is SWI evidence of abnormal susceptibility artifact associated with intraaxial blood products. [Yes/No]
There is SWI evidence of abnormal any susceptibility artifact associated with intraventricular or subependymal blood products. [Yes/No]
There is SWI evidence of abnormal susceptibility artifact associated with subpial spaces related to trauma. [Yes/No]
There is SWI evidence of parenchymal linear susceptibility artifact following a cortical vein consistent with venous tether injury and resultant brain laceration. [Yes/No]
There is SWI evidence of a few, multiple, or even diffuse parenchymal punctate susceptibility artifacts, especially in the subpial spaces, gray-white matter junction, or major forceps corpus callosum consistent with DAI. [Yes/No]
There is SWI evidence of a few or multiple or even diffuse parenchymal punctate susceptibility artifacts, especially in the corpus callosum, basal ganglia, thalamus, mesencephalon and central brain stem consistent with end-artery vascular shear injuries associated with acceleration-deceleration injuries. [Yes/No]
There is SWI evidence of thrombus within the lumen (exhibiting blooming artifact) of any major cortical vein or dural sinus. [Yes/No]
There is altered SWI appearance of the venocapillary pool and the deep venous system, either increased from dysautoregulation-related hyperemia or reduced from vasospasm. [Yes/No]
Other
There is evidence of concurrent abnormalities not likely related to recent brain injury. [Yes/No]